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Link: Steroid-Responsive Encephalopathy Associated with Autoimmune Thyroiditis Presenting with Diffusion MR Imaging Changes

Steroid-responsive encephalopathy associated with autoimmune thyroiditis (SREAT) presents with focal or diffuse nonenhancing MR imaging abnormalities in 50% of patients with SREAT during subacute exacerbation. Vasculitic changes in biopsy studies as well as the elevation of antithyroid antibodies and CSF protein suggests an inflammatory cause. We report the case of a patient with SREAT with changes on diffusion-weighted MR imaging, which improved with corticosteroid therapy and plasmapheresis, supporting the theory of inflammatory changes in exacerbation of presumptive SREAT.

The full text of this study can be found at http://www.ajnr.org/content/29/8/1550.full.

Copyright © American Society of Neuroradiology | February 17, 2008
by C. Grommesa, C. Griffina, K.A. Downesb and A.J. Lernera

This website is not a substitute for independent professional advice. Nothing contained in this site is intended to be used as medical advice. No articles, personal accounts, or other content are intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professionals advice.

Link: Hashimoto’s Encephalopathy Complicated by Cerebellar Ataxia: A Case Report

OBJECTIVE

To present a case of Hashimoto’s Encephalopathy (HE) with long-term neurological sequelae.

BACKGROUND:

Hashimoto’s Encephalopathy, also known as steroid responsive encephalopathy secondary to autoimmune thyroiditis (SREAT), is a rare condition that typically affects middle aged females with known anti-TPO antibodies and a history of Hashimoto’s thyroiditis. The diagnosis is often overlooked because there is no diagnostic biomarker, and patients can present with several non-specific neurological symptoms that are often incorrectly attributed to other disease processes. Neurological outcomes are rarely reported. It is important to recognize and distinguish HE from other encephalopathies as it is a distinct, treatable entity, which is almost always responsive to steroid therapy.

The study can be found at http://www.neurology.org/content/82/10_Supplement/P6.023.short. A membership may be needed to read the full text.

Copyright © 2014 American Academy of Neurology | by Dr. Michael Hossack and Dr. Perry Richardson

 

 

 

 

This website is not a substitute for independent professional advice. Nothing contained in this site is intended to be used as medical advice. No articles, personal accounts, or other content are intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professionals advice.

Link: A follow-up 18F-FDG brain PET study in a case of Hashimoto’s encephalopathy causing drug-resistant status epilepticus treated with plasmapheresis

Hashimoto’s encephalopathy (HE) is a rare neuropsychiatric syndrome associated with antithyroid antibodies. It may have an acute onset (episodes of cerebral ischemia, seizure, and psychosis) or it may present as an indolent form (depression, cognitive decline, myoclonus, tremors, and fluctuations in level of consciousness). We here describe a case of encephalopathy presenting as non-convulsive status epilepticus associated with Hashimoto’s thyroiditis (HT), unresponsive to corticosteroid therapy, with improvement after plasma exchange treatment.

The article appears in the Journal of Neurology and the text can be found at http://link.springer.com/article/10.1007/s00415-013-7228-0#page-1. However, you may have to purchase the article or log into the site to read the full text.

 

Copyright © Springer-Verlag Heildelberg 2014 | Journal of Neurology | April 2014, Volume 261, Issue 4, pp 663-667

This website is not a substitute for independent professional advice. Nothing contained in this site is intended to be used as medical advice. No articles, personal accounts, or other content are intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professionals advice.